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"I wouldn't be surprised if researchers and labs are looking for ways to activate SLC25A51 chemically to get the NAD+ flowing to SIRT3 without requiring fasting."

To what end ?

Fasting would be cheaper, easier and less time-consuming and would include all of the related, broad-spectrum effects that we also believe fasting may produce.

I guess if you're a pharma investor or existing, entrenched player it makes sense to go that direction.

As a consumer, however, I wonder why (pills that might convey a subset of the benefits of fasting) would be interesting in any way.



> As a consumer, however, I wonder why

Because fasting is hard?




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